Health & Medical · Cardiovascular reference
Cholesterol Level by Age Chart for Children and Adults
Compare cholesterol levels across childhood, adolescence, adulthood, and older age. Review LDL, HDL, non-HDL, triglycerides, screening windows, current risk-based LDL goals, and inherited high-cholesterol warning signs.
Do not choose a treatment target from age alone. Pregnancy, childhood, diabetes, kidney disease, coronary calcium, known cardiovascular disease, and cholesterol medicines require professional interpretation. Read the ChartsLoom Disclaimer.
What cholesterol level is healthy for your age?
For people age 19 or younger, desirable levels include total cholesterol below 170 mg/dL, LDL below 110 mg/dL, non-HDL below 120 mg/dL, and HDL above 45 mg/dL. For most adults, broad desirable levels include total cholesterol below 200 mg/dL, LDL below 100 mg/dL, and non-HDL below 130 mg/dL.
These values describe general screening ranges, not a universal treatment goal. The 2026 ACC/AHA dyslipidemia guideline summary uses risk-based LDL goals: below 100 mg/dL for many adults at borderline or intermediate risk, below 70 mg/dL at high risk, and below 55 mg/dL for very-high-risk secondary prevention.
- Pediatric LDL
- <110 mg/dL
- This is the acceptable LDL cut point for people age 19 or younger.
- General adult LDL
- <100 mg/dL
- Higher-risk adults may have goals below 70 or even 55 mg/dL.
- Child screening
- Ages 9–11
- Universal screening is recommended once during this window.
- Possible FH
- LDL ≥190 adult
- Very high LDL can signal an inherited cholesterol disorder.
Cholesterol interpretation by age and life stage
Age changes which screening pathway and risk model clinicians use. It does not create a separate “normal” adult LDL range for every decade.
Swipe horizontally inside the table to view every column.
| Age or life stage | Usual screening approach | How results are interpreted | Key point |
|---|---|---|---|
| Birth to 1 year | No routine universal lipid screening | Testing is reserved for unusual clinical or family-risk situations | Infants do not use ordinary child or adult cholesterol cutoffs |
| Age 2–8 years | Selective testing when family history or major risk conditions are present | Pediatric lipid cut points | Earlier testing may be appropriate with familial hypercholesterolemia or major risk factors |
| Age 9–11 years | Universal screening once if not already completed — Universal childhood screening window | Pediatric total, LDL, non-HDL, HDL, and triglyceride cut points | A nonfasting panel may identify who needs a fasting profile |
| Age 12–16 years | Selective repeat testing when risk factors, family history, or prior abnormalities are present | Pediatric cut points remain in use | Puberty can temporarily shift lipid levels |
| Age 17–21 years | Universal screening once during this period | Transition from pediatric to adult risk-based care | The laboratory and clinician determine which framework applies at the boundary |
| Age 19–39 years | Obtain a complete lipid panel and repeat based on risk | General adult levels plus lifetime risk, family history, and inherited conditions | Early LDL exposure matters even when short-term risk is low |
| Age 40–75 years | Routine panels with risk reassessment as clinically indicated | PREVENT-ASCVD risk, diabetes, kidney disease, smoking, blood pressure, and risk enhancers guide treatment — Risk-based adult treatment period | Age affects cardiovascular risk, not the basic meaning of LDL |
| Age 76 years and older | Continue individualized testing | Prior ASCVD, frailty, life expectancy, medicines, goals, and tolerance shape decisions | Treatment is individualized rather than stopped solely because of age |
| Pregnancy and lactation | Test when clinically indicated | Interpret expected physiologic changes and defer most lipid-lowering medicines unless a specialist advises otherwise | Do not change prescribed therapy without obstetric and cardiovascular guidance |
Ages are chronological years. Screening timing can be earlier or more frequent when risk is higher.
- • Adult LDL goals are based on cardiovascular risk and disease history, not on a decade-by-decade “normal” range.
- • Children and adolescents use pediatric cut points because lipid distributions and treatment pathways differ from adults.
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How cholesterol interpretation changes across the lifespan
The measurement stays the same, but the screening pathway and treatment framework change. Childhood uses population cut points; adulthood increasingly uses individual cardiovascular risk.
Age 2–19
Childhood
Use pediatric total, LDL, non-HDL, HDL, and age-specific triglyceride cut points.
Age 19–39
Young adulthood
Identify inherited high LDL early and consider lifetime exposure, family history, and Lp(a).
Age 40–75
Midlife risk assessment
Combine the lipid panel with PREVENT-ASCVD risk, diabetes, kidney disease, blood pressure, and smoking.
Age 76+
Older adulthood
Continue individualized prevention based on ASCVD, health status, frailty, medicines, and goals.
Direct answer
Cholesterol often rises with age, but a higher LDL does not become healthy simply because a person is older. Adult goals are based on the probability of heart attack or stroke and can become lower as risk increases.
Pediatric cholesterol levels for ages 19 and younger
These pediatric cut points classify screening results as acceptable, borderline, or high. A clinician confirms abnormal results and considers age, puberty, family history, weight, diabetes, kidney disease, and medicines.
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| Lipid measurement | Acceptable | Borderline | High or low concern |
|---|---|---|---|
| Total cholesterol | <170 mg/dL | 170–199 mg/dL | ≥200 mg/dL |
| LDL cholesterol | <110 mg/dL — Acceptable pediatric LDL | 110–129 mg/dL | ≥130 mg/dL |
| Non-HDL cholesterol | <120 mg/dL | 120–144 mg/dL | ≥145 mg/dL — High pediatric non-HDL |
| HDL cholesterol | >45 mg/dL | 40–45 mg/dL | <40 mg/dL is low |
| Triglycerides, age 0–9 | <75 mg/dL | 75–99 mg/dL | ≥100 mg/dL |
| Triglycerides, age 10–19 | <90 mg/dL | 90–129 mg/dL | ≥130 mg/dL |
| Apolipoprotein B | <90 mg/dL | 90–109 mg/dL | ≥110 mg/dL |
To convert total, LDL, HDL, or non-HDL cholesterol from mg/dL to mmol/L, divide by 38.67. For triglycerides, divide by 88.57.
- • A screening result is not the same as a treatment target. Persistent abnormalities usually require repeat testing and complete risk assessment.
- • Triglyceride cut points differ for children younger than 10 and those ages 10–19.
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Children do not use adult cholesterol categories
The NHLBI pediatric lipid guidance uses lower total, LDL, non-HDL, and triglyceride cut points. Persistent high LDL needs repeat testing and complete family-risk assessment rather than an adult calculator.
General adult cholesterol levels from age 20 onward
These are broad desirable levels for many adults. Personal LDL and non-HDL goals may be substantially lower when cardiovascular risk, diabetes, kidney disease, coronary calcium, or established ASCVD is present.
Swipe horizontally inside the table to view every column.
| Lipid measurement | General desirable level | Higher-risk context | What the number represents |
|---|---|---|---|
| Total cholesterol | <200 mg/dL | ≥240 mg/dL has traditionally been considered high | Combined cholesterol carried in several lipoproteins |
| LDL cholesterol | <100 mg/dL — General desirable adult LDL | ≥190 mg/dL suggests severe hypercholesterolemia and possible inherited disease | Primary cholesterol carried in atherogenic LDL particles |
| Non-HDL cholesterol | <130 mg/dL | Risk-based goals can be lower | Total cholesterol minus HDL; includes LDL and other atherogenic particles |
| HDL cholesterol, men | ≥60 mg/dL is favorable; <40 mg/dL is low | High HDL does not cancel high LDL or other risk | Cholesterol carried in HDL particles |
| HDL cholesterol, women | ≥60 mg/dL is favorable; <50 mg/dL is low | Menopause and metabolic risk can change the pattern | Cholesterol carried in HDL particles |
| Triglycerides | <150 mg/dL | ≥500 mg/dL requires prompt clinical assessment because pancreatitis risk rises — Severe triglyceride concern | Circulating storage fat influenced by meals, alcohol, diabetes, and genetics |
| Lipoprotein(a) | No universal “optimal” value; measure at least once in adulthood | ≥125 nmol/L or ≥50 mg/dL is a risk-enhancing level | Mostly inherited cholesterol-carrying particle |
Cholesterol is usually reported in mg/dL in the United States and mmol/L in many other countries.
- • The 2026 guideline restores risk-based LDL and non-HDL goals rather than relying only on descriptive categories.
- • A healthy HDL value does not make an elevated LDL level harmless.
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Adult goals depend on risk, not birthday alone
The American Heart Association cholesterol overview explains that a lipid panel must be combined with personal risk. Two adults of the same age can have different LDL goals because their cardiovascular histories and risk factors differ.
2026 LDL goals by cardiovascular risk
The current guideline uses age, PREVENT-ASCVD risk, diabetes, kidney disease, coronary calcium, prior cardiovascular disease, and other risk enhancers to select an LDL goal.
Swipe horizontally inside the table to view every column.
| Clinical context | Common LDL goal | Additional context | What age contributes |
|---|---|---|---|
| Low primary-prevention risk | No single medication target for everyone | Lifestyle and periodic reassessment remain central | Age contributes to lifetime and short-term risk |
| Borderline or intermediate primary-prevention risk | <100 mg/dL — Borderline or intermediate risk LDL goal | Treatment depends on shared decision-making and risk enhancers | PREVENT-ASCVD applies to adults ages 30–79 without known ASCVD |
| High primary-prevention risk | <70 mg/dL | More intensive lowering may be appropriate | Age is one part of the risk estimate |
| Any coronary artery calcium | <100 mg/dL, with lower goals as calcium burden rises | CAC can clarify decisions when risk remains uncertain | CAC is selectively considered in adults, commonly men ≥40 and women ≥45 |
| Established ASCVD, not very high risk | At least <70 mg/dL | Secondary prevention generally needs intensive therapy | Chronological age does not remove the benefit of risk reduction |
| Established ASCVD, very high risk | <55 mg/dL — Very high risk secondary-prevention LDL goal | Non-HDL goal <85 mg/dL | Goal is based on event risk rather than age alone |
| LDL ≥190 mg/dL | Prompt evaluation and intensive lowering | Consider familial hypercholesterolemia and cascade screening | Can occur in children, young adults, and older adults |
| Age >75 without known ASCVD | Individualized | Consider health status, frailty, life expectancy, interactions, and preferences | Therapy may still be considered with lifestyle measures |
LDL-C goals are in mg/dL. Divide by 38.67 for mmol/L.
- • These are guideline goals, not self-treatment instructions. Medication decisions require a clinician-patient discussion.
- • The same LDL value can lead to different recommendations in two people because their total cardiovascular risk differs.
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Triglyceride levels and age-related context
Triglycerides often rise with insulin resistance, weight gain, alcohol intake, some medicines, kidney or thyroid disease, and age-related metabolic changes. The thresholds themselves do not increase simply because someone is older.
Swipe horizontally inside the table to view every column.
| Triglyceride result | General adult category | Why it matters | Typical next step |
|---|---|---|---|
| <150 mg/dL — Usual adult triglyceride level | Usually considered normal | Lower pancreatitis and metabolic-risk concern | Interpret with LDL, non-HDL, HDL, glucose, and overall risk |
| 150–199 mg/dL | Borderline high or elevated | Can be a risk-enhancing metabolic marker | Review alcohol, refined carbohydrates, weight, diabetes control, and medicines |
| 200–499 mg/dL | High | Atherogenic remnant particles and metabolic risk become more important | Clinical evaluation and treatment based on ASCVD risk and causes |
| ≥500 mg/dL — Severe triglyceride level | Severe | Pancreatitis risk becomes a major treatment concern | Prompt clinician-guided treatment and secondary-cause review |
| ≥1,000 mg/dL | Markedly severe | Pancreatitis risk can be substantial | Urgent specialist-directed management |
| Pregnancy | Can rise physiologically | Very high values can still be dangerous | Obstetric and lipid-specialist interpretation |
| Child age 0–9 | High begins at ≥100 mg/dL | Pediatric cut point is lower than the adult level | Use pediatric evaluation and repeat testing |
| Age 10–19 | High begins at ≥130 mg/dL | Puberty, obesity, diabetes, and family history affect context | Use pediatric evaluation and repeat testing |
For triglycerides, mmol/L = mg/dL ÷ 88.57.
- • A nonfasting sample can still be useful, but a clinician may request a fasting repeat when triglycerides are elevated.
- • Severe abdominal pain, vomiting, or a very high triglyceride result requires prompt medical assessment.
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Cholesterol result checker by age
Enter a lipid-panel result to apply pediatric or general adult descriptive cut points and calculate non-HDL cholesterol. This tool does not calculate PREVENT-ASCVD risk or recommend treatment.
Cholesterol screening schedule by age and risk
Screening intervals are starting points. Prior abnormal results, treatment, pregnancy, diabetes, kidney disease, family history, and cardiovascular disease can require earlier or more frequent testing.
Swipe horizontally inside the table to view every column.
| Age or circumstance | General schedule | Reasons to test sooner | Test type or follow-up |
|---|---|---|---|
| Age 2–8 | Selective screening | Parent with early ASCVD, known familial hypercholesterolemia, diabetes, hypertension, obesity, kidney disease, or other high-risk condition | Nonfasting or fasting panel according to clinician plan |
| Age 9–11 | Universal screening once — Universal child screening window | Earlier abnormal result or major risk condition | Non-HDL screening; fasting confirmation when needed |
| Age 12–16 | Selective screening | New risk factors, family history, or prior abnormal result | Repeat panel and complete risk review |
| Age 17–21 | Universal screening once | Known dyslipidemia or treatment monitoring | Transition toward adult care |
| Age 19–39 | About every 5 years for many adults | Family history, LDL elevation, diabetes, high blood pressure, smoking, obesity, kidney disease, inflammatory disease, or pregnancy-related risk history | Complete lipid panel; consider Lp(a) once in adulthood |
| Men 45–65 and women 55–65 | Often every 1–2 years | Any abnormal result or treatment change | Repeat lipid panel and risk reassessment |
| Age >65 | Often annually | Treatment changes, cardiovascular disease, frailty, illness, or medication interactions | Individualized interval |
| After starting or changing lipid-lowering therapy | Clinician-specified interval | Assess response, adherence, and adverse effects | Repeat LDL and related laboratory tests |
| Strong family history or suspected FH | Do not wait for the routine window | LDL ≥190 mg/dL in adults or ≥160 mg/dL in children, tendon findings, or early family heart disease — Familial hypercholesterolemia warning | Prompt evaluation and family cascade screening |
Intervals are approximate general recommendations and do not replace a personal follow-up plan.
- • The 2026 guideline recommends a complete panel for adults age 19 and older and universal screening for children ages 9–11 if not previously screened.
- • An Lp(a) measurement is recommended at least once in adulthood because the value is largely inherited and stable.
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Screening frequency increases when risk rises
The MedlinePlus age-based testing guidance recommends universal testing in childhood and periodic adult testing, with shorter intervals for middle-aged and older adults or anyone with abnormal results and cardiovascular risk factors.
What each lipid-panel value means
A lipid panel reports several related values. Clinicians interpret the pattern rather than treating total cholesterol as the only meaningful number.
Swipe horizontally inside the table to view every column.
| Test or calculation | How it is obtained | Main use | Important limitation |
|---|---|---|---|
| Total cholesterol | Measured | Broad screening summary | Can look acceptable even when LDL or non-HDL is high |
| HDL cholesterol | Measured | Risk context and non-HDL calculation | High HDL does not neutralize high LDL |
| Triglycerides | Measured | Metabolic and pancreatitis-risk context | Can change after food, alcohol, illness, or poor glucose control |
| LDL cholesterol | Calculated or directly measured | Primary atherogenic-cholesterol treatment marker | Calculated LDL can be less reliable with very high triglycerides |
| Non-HDL cholesterol | Total cholesterol − HDL — Non-HDL formula | Captures cholesterol in all atherogenic particles | Still requires risk-based interpretation |
| Apolipoprotein B | Measured separately | Estimates the number of atherogenic particles | Not included in every standard panel |
| Lipoprotein(a) | Measured separately | Inherited risk-enhancing marker — Inherited Lp(a) risk marker | Lifestyle changes have little effect on the level |
| Coronary artery calcium | CT imaging, not a blood test | Clarifies subclinical plaque burden when treatment is uncertain | Not a routine test for everyone |
| PREVENT-ASCVD risk | Calculator using age and clinical factors | Estimates 10- and 30-year risk for eligible adults | Not a cholesterol value and not used in people with known ASCVD |
Non-HDL cholesterol = total cholesterol − HDL cholesterol.
- • A fasting sample is not required for every routine panel, but it may be requested when triglycerides are high or a calculated LDL needs confirmation.
- • Age contributes to risk calculators because cumulative exposure and event risk rise over time.
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Why cholesterol can change with age
Age-related patterns often reflect cumulative lifestyle exposure, menopause, changes in muscle and body fat, chronic disease, medicines, and genetics rather than age alone.
Swipe horizontally inside the table to view every column.
| Factor | Possible lipid effect | Age-related context | What to review |
|---|---|---|---|
| Genetics and familial hypercholesterolemia | Markedly high LDL from childhood onward — Inherited high LDL pattern | Can cause premature ASCVD across generations | Family history, repeat LDL, physical findings, and genetic evaluation |
| Puberty | Temporary shifts in total and LDL cholesterol | Can affect adolescent screening values | Repeat abnormal results according to pediatric guidance |
| Menopause | LDL and non-HDL may rise | Hormonal changes alter lipid metabolism — Menopause-related context | Overall cardiovascular risk and treatment options |
| Weight gain and central adiposity | Triglycerides and non-HDL may rise; HDL may fall | Body composition often changes with age | Weight trend, waist, glucose, blood pressure, and activity |
| Diabetes or insulin resistance | High triglycerides, low HDL, and atherogenic particles | Prevalence rises with age but can occur in youth | A1C, glucose control, kidney function, and medicines |
| Hypothyroidism | LDL and triglycerides may rise | More common with increasing age | Thyroid testing when clinically indicated |
| Kidney or liver disease | Can alter multiple lipid fractions | Chronic conditions become more common over time | Underlying disease and treatment plan |
| Medicines | Can raise or lower LDL, HDL, or triglycerides | Medication burden often increases with age | Steroids, hormones, retinoids, antipsychotics, HIV therapy, and others |
| Alcohol and refined carbohydrates | Triglycerides may rise | Tolerance and metabolic effects can change with age | Intake pattern and liver health |
| Acute illness or recent major weight change | Temporary lipid shifts | Can make one result unrepresentative | Repeat testing after recovery when advised |
Direction and magnitude vary by person and condition.
- • High cholesterol usually causes no symptoms, so age-related risk cannot be judged by how someone feels.
- • Lifestyle changes help, but inherited disorders often require medication in addition to healthy habits.
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Familial hypercholesterolemia warning chart
Familial hypercholesterolemia is an inherited condition that causes very high LDL from early life. Prompt recognition allows earlier treatment and testing of relatives.
Swipe horizontally inside the table to view every column.
| Finding | Why it raises concern | Age context | Recommended response |
|---|---|---|---|
| Adult LDL >190 mg/dL — Adult FH warning level | Major sign of possible FH | Can be detected at any adult age | Prompt clinical evaluation and repeat confirmation |
| Child LDL >160 mg/dL — Child FH warning level | Possible inherited hypercholesterolemia | Particularly important with family history | Pediatric lipid-specialist evaluation |
| Heart attack or coronary disease at a young age in a close relative | Suggests inherited premature ASCVD risk | Family history often predates the patient’s symptoms | Earlier screening and family cascade testing |
| Tendon xanthomas or painful, thickened Achilles tendons | Can reflect cholesterol deposition | May become more visible with time | Clinical examination and specialist referral |
| Yellowish deposits around the eyes | Possible cholesterol deposition | Not specific for FH | Interpret with LDL and family history |
| Corneal arcus at an unusually young age | Can accompany inherited high LDL | More concerning when premature | Clinical assessment |
| One parent with confirmed FH | Autosomal dominant inheritance creates substantial child risk | Screen children early | Coordinate family testing |
| Healthy lifestyle but LDL remains very high | Genetic LDL clearance problem may be present | Diet alone is often insufficient | Medication is commonly required |
| Known FH plus chest pain, pressure, fainting, or stroke symptoms | Possible acute cardiovascular event | Emergency risk can occur earlier than usual | Call emergency services |
LDL thresholds are screening clues, not standalone genetic diagnoses.
- • The CDC identifies LDL above 190 mg/dL in adults and above 160 mg/dL in children as major FH warning signs.
- • Relatives may need testing even when they feel healthy because elevated LDL usually has no symptoms.
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Very high LDL can be inherited
The CDC familial hypercholesterolemia guide identifies LDL above 190 mg/dL in adults and above 160 mg/dL in children as major warning signs. Family members may need testing even when they feel healthy.
Frequently asked questions
What is a healthy cholesterol level by age?
For people age 19 or younger, desirable levels include total cholesterol below 170 mg/dL, LDL below 110 mg/dL, non-HDL below 120 mg/dL, and HDL above 45 mg/dL. For most adults, general desirable levels include total cholesterol below 200 mg/dL, LDL below 100 mg/dL, and non-HDL below 130 mg/dL, but personal goals may be lower when cardiovascular risk is high.
Does normal LDL cholesterol increase with age?
No. LDL often rises as people age, but that does not make a higher result healthy. Adult LDL goals are selected by cardiovascular risk, disease history, diabetes, kidney disease, coronary calcium, and other factors rather than by accepting a higher decade-specific normal.
At what age should a child have cholesterol testing?
Universal childhood screening is recommended once between ages 9 and 11. Earlier selective testing may begin around age 2 when familial hypercholesterolemia, premature family heart disease, diabetes, obesity, kidney disease, or another major risk factor is present.
When should teenagers be screened again?
A second universal screening window is recommended between ages 17 and 21. Testing may occur sooner during adolescence when a prior result was abnormal or new risk factors appear.
What LDL goal applies to adults at high cardiovascular risk?
The 2026 guideline uses an LDL goal below 70 mg/dL for high-risk primary prevention. People with established ASCVD at very high risk generally have an LDL goal below 55 mg/dL. A clinician determines the correct category.
What cholesterol number suggests familial hypercholesterolemia?
An LDL above 190 mg/dL in an adult or above 160 mg/dL in a child is a major warning sign, especially with a family history of early heart disease. These values require clinical evaluation rather than self-diagnosis.
Is HDL of 60 mg/dL always protective?
An HDL level of 60 mg/dL or higher is generally favorable, but it does not cancel an elevated LDL, high non-HDL, diabetes, smoking, high blood pressure, or other risk factors.
What is non-HDL cholesterol?
Non-HDL cholesterol equals total cholesterol minus HDL cholesterol. It includes LDL and other cholesterol carried in atherogenic particles, making it useful when triglycerides are elevated or several particle types contribute to risk.
Do cholesterol tests require fasting?
Not every routine cholesterol panel requires fasting. A clinician may request a fasting repeat when triglycerides are elevated, a calculated LDL is uncertain, or a treatment decision needs a standardized measurement.
How often should adults check cholesterol?
Many younger adults with low risk are tested about every five years. Testing often becomes more frequent during middle age and after age 65, and it occurs sooner when cholesterol is abnormal, treatment changes, or cardiovascular risk is high.
Why does cholesterol often rise after menopause?
Hormonal changes after menopause can raise LDL and non-HDL cholesterol and alter fat distribution. The result should be interpreted with blood pressure, glucose, smoking, kidney health, family history, and overall cardiovascular risk.
What triglyceride level is dangerous?
Triglycerides at 500 mg/dL or higher require prompt clinical assessment because pancreatitis risk becomes more important. Levels around 1,000 mg/dL or higher can carry substantial risk and may need urgent specialist treatment.
Can children have high cholesterol without obesity?
Yes. Familial hypercholesterolemia and other inherited disorders can cause very high LDL in children who have a healthy weight and active lifestyle. Family history and universal screening help identify these cases.
Can I use one cholesterol result to decide whether I need medicine?
No. Treatment decisions use repeat measurements when needed, age, cardiovascular history, PREVENT-ASCVD risk, diabetes, kidney disease, family history, pregnancy status, coronary calcium, risk enhancers, and personal preferences.
Sources
These organizations provide the lipid cut points, screening recommendations, current risk-based goals, and inherited-cholesterol context used on this page.
American Heart Association and American College of Cardiology
2026 Guideline on the Management of Dyslipidemia
Current risk-based LDL goals, PREVENT-ASCVD use, childhood screening, Lp(a), coronary calcium, and treatment context.
https://newsroom.heart.org/news/accaha-issue-updated-guideline-for-managing-lipids-cholesterol
MedlinePlus, U.S. National Library of Medicine
Cholesterol Levels: What You Need to Know
General desirable cholesterol levels for people age 19 or younger and adults age 20 or older, plus screening frequency.
https://medlineplus.gov/cholesterollevelswhatyouneedtoknow.html
National Heart, Lung, and Blood Institute
Integrated Guidelines for Cardiovascular Health in Children and Adolescents
Pediatric acceptable, borderline, and high cut points for total, LDL, non-HDL, HDL, and triglycerides.
https://www.nhlbi.nih.gov/files/docs/guidelines/peds_guidelines_full.pdf
American Heart Association
What Your Cholesterol Levels Mean
Adult screening intervals, triglyceride context, and the principle that personal goals depend on total cardiovascular risk.
https://www.heart.org/en/health-topics/cholesterol/about-cholesterol/what-your-cholesterol-levels-mean
Centers for Disease Control and Prevention
About Familial Hypercholesterolemia
Familial hypercholesterolemia warning levels, inheritance context, family screening, and early treatment importance.
https://www.cdc.gov/heart-disease-family-history/about/about-familial-hypercholesterolemia.html